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Interestingly, theseLFS mouse models exhibit variable phenotypes consistent with LFS in humans.A model containing the Trp53R172H mutation exhibited increased metastatic poten-tial compared to Trp53+/? mice even though levels of the mutated p53 protein wereas low as those observed in wildtype p53 mice due to a splicing abnormality [ 42 ].These ?ndings demonstrate that potent gain-of-function effects of missense Trp53mutations can signi?cantly alter phenotype even when expressed at low levels.Moreover, Trp53R172H/+ mice showed a signi?cant increase in the number of carcino-mas and a decrease in the number of lymphomas compared to Trp53 +/? mice.
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